Endometrial Hyperplasia - Knowing Your Own Diagnosis

Endometrial Hyperplasia is a disorder that is believed to be one of the pre-disposing factors for the development of uterine cancer. This disorder is brought by different hormonal alterations in the body. The public views this disorder as cancer itself or a developing uterine anomaly. Most of us are not yet aware about how diagnoses are read and interpreted.

Endometrial Hyperplasia involves alteration between two hormones. Estrogen is the hormone responsible for the development of female sex characteristics. It has a growth-stimulating factor that induces the thickening of the endometrial wall. It induces hyperplasia or cellular proliferation in order to prepare the endometrium for possible conception. The other hormone, progesterone, works as its regulator. Progesterone regulates the phase of cellular division in order to prevent excessive endometrial thickening. If conception does not occur, the endometrium sheds off in a form of menstruation.

The condition of Endometrial Hyperplasia occurs if the production of progesterone hormone becomes altered. If progesterone levels drop, the cellular division increases due to the absence of regulation. This excessively thickens the endometrial wall because of the continuous induction of estrogen. If this situation is prolonged, further complication may develop. Atypia or cellular abnormality arises if cells are exposed to too much levels of estrogen for longer periods. The diagnostic procedures for this disorder are vastly available in almost all hospital setting. The common problem is the understanding and interpretation of these diagnoses.

There are different grades to be understood in terms of endometrial hyperplasia. The common diagnoses that the health care providers give are as follows.

Relatively Benign. This diagnosis depicts Endometrial Hyperplasia but most likely belongs to simple to complex without atypia category. This is has the least risk among all the diagnosis. The chances for uterine cancer progression are very low.

Probable Benign. The hormone stimulation is present and cellular division is active. The hormone alteration is probably taking place. This condition is also less risky but medical follow up and continuous monitoring is necessary. This condition is usually alleviated by simple hormonal therapy.

Precancerous changes and cellular aggressiveness is observed. This type of diagnosis is very risky and requires immediate attention for further prevention. This stage is commonly experiencing a complex hyperplasia with evident atypia or abnormality. If this condition is not resolved, progression to uterine cancer might occur.

Cancerous. This diagnosis is the most dangerous of all. It indicates positive cancer cells proliferating in the uterine cavity. This requires immediate oncology management for treatment and prevention for its further progression. Such conditions usually lead to Atypical Adenomatous Hyperplasia, Endometrial Adenocarcinoma, Adenocanthoma, Endometrial intraepithelial neoplasia and invasive carcinoma.

These conditions are malignant and may require the patient to undergo hysterectomy or the removal of the whole uterus in order to prevent possible spread. This condition is very devastating for most of the females because their capacity for conception is greatly at risk. Knowing the interpretations of different diagnoses can serve as the life saving knowledge for us. This leads to early prevention which is primarily the most effective treatment for health anomalies especially cancer.

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